[It is a technical point unnecessary to raise whether the adhesions
formed in the first or the last period; they were formed without
question; I and if they were formed in the beginning, as doubtless
they were, they withstood the most severe and trying period of their
existence, which was before the abscess broke into the bowels, and
so far as being able to resist to the very last, there has been no
evidence to prove that the last infection was because of any lack of
power of resistance on their part for the autopsy showed them
intact. It is doubtful if anything but sound tissue could have
withstood the strain that was put upon this man's diseased cecum
from gas distention. The infection-laden gas could find a way
anywhere in diseased tissue and broken continuity. Why should the
pus break through the adhesions and find its way into the peritoneum
after they had been able to make an effectual resistance till the
bulk of it had forced a passage into the bowel? Why should the
adhesions have less power to resist when there is less strain upon
them and also a patent outlet for the pus? I fear our German friend
of "Die Deutsche Klinik" had "booze" in his logic when he was
explaining how his patient came to die.]

"Moreover, the bacterial finding of streptococci and cold bacilli in
the perityphlitic abscess is typical, and the limitation of the
diffuse peritonitis to areas below the omentum is also instructive.
This simultaneously prevented the invasion of organisms producing
inflammation into the serous surfaces above."

[There is nothing strange about this for nature works for the
purpose of preventing "serous surface" invasion, and it takes a
deal of malpractice to force such an infection. If nature's
provisions against peritoneal inflammation were not as great as they